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A mathematical model to quantify the effects of platelet count, shear rate, and injury size on the initiation of blood coagulation under venous flow conditions dynamics is formulated. The model predicts that a high platelet count increases the peak value of generated

Platelet activation in heparin-induced thrombocytopenia is followed by platelet death via complex apoptotic and non-apoptotic pathways-apoptotic pathways that may underlie the low platelet count in HIT.

Blood platelets in the development of sepsis, septic shock and multiple organ failure syndrome develops rapidly, with minimal platelet counts registered on the fourth day of observation, after which

Clinical efficacy and safety of nebulized prostacyclin in patients with sARs-CoV-2 (prospective comparative study) [клиНичеСкая ЭффективНоСть и безоПаСНоСть ПримеНеНия иНГаляциоННоГо ПроСтациклиНа у больНых С иНфекцией, вызваННой SARS-COV-2 (ПроСПективНое СравНительНое иССледоваНие)] and platelet counts were significantly higher in control group We have also noticed significantly lower level

Intracellular origin and ultrastructure of platelet-derived microparticles© 2017 International Society on Thrombosis and Haemostasis Essentials Platelet microparticles play

Coronavirus disease (COVID-19) and disseminated intravascular coagulation [Коронавирусная инфекция (COVID-19) и синдром диссеминированного внутрисосудистого свертывания]-dimer level, prothrombin time, platelet count and fibrinogen content is important for determining indications

Kinetics and mechanics of clot contraction are governed by the molecular and cellular composition of the blood+, the integrin αIIbβ3, myosin IIa, FXIIIa cross-linking, and platelet count all promote 1 or more phases

Quantitative and qualitative changes in blood cells associated with COVID-19 counts and morphology along with the platelet function by flow cytometry in comparison with the clinical

COVID-19 associated coagulopathy: The crowning glory of thrombo-inflammation conceptplatelet count

Pleyotropic antiaggregant effects of an innovative antiarrhythmic of class III SS-68, an indole derivative an antiaggregant activity. The influence of SS-68 on the platelet formation stage of hemostasis was determined

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