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Cooperative hydrogen bonding in solution: Influence of molecule structure⋯ROH⋯B (B-base), CCl3H⋯ROH⋯O(C2H5)2 and CCl3H⋯ROH⋯ROH in proton donor solvent CCl3H were studied. Solvent

Multilocus association analysis identifies genetic predictors of essential hypertension8, CCL16, CCL17, CCL18, CXCL1, CXCL8, CXCL13, CCR2, CCR5, CXCR2, CXCR4, CX3CR1, CCL23) in the group

Elevated levels of proinflammatory cytokines in cerebrospinal fluid of multiple sclerosis patients cytokines, including IL-2RA, CCL5, CCL11, MIF, CXCL1, CXCL10, IFNγ, SCF, and TRAIL, were upregulated

Cooperative hydrogen bonding in solution: Influence of molecule structure⋯ROH⋯B (B-base), CCl3H⋯ROH⋯O(C2H5)2 and CCl3H⋯ROH⋯ROH in proton donor solvent CCl3H were studied. Solvent

CHEMOKINE GENE POLYMORPHISMS ASSOCIATION WITH INCREASED RISK OF TYPE 2 DIABETES MELLITUS IN TATAR ETHNIC GROUP, RUSSIA [odds ratio (OR) = 2.77 (95% CI 1.81–4.25), р = 0.0001], rs2107538 in CCL5 [odds ratio (OR) = 1.80 (95

CHEMOKINE GENE POLYMORPHISMS ASSOCIATION WITH INCREASED RISK OF TYPE 2 DIABETES MELLITUS IN TATAR ETHNIC GROUP, RUSSIA [odds ratio (OR) = 2.77 (95% CI 1.81–4.25), р = 0.0001], rs2107538 in CCL5 [odds ratio (OR) = 1.80 (95

Generating phosphinidene - N-methylimidazole adducts under mild conditions, this reaction effectively generates the [PhP] adduct, which decomposes to give essentially Ph 4P 4 and Ph 5P 5

The skin-brain connection hypothesis, bringing together CCL27-mediated T-cell activation in the skin and neural cell damage in the adult brain these lymphocytes may initiate an inflammatory response. Our observation of increased CC chemokine ligand 27 (CCL27

Evaluation of the Hepatoprotective Effect of L-Ascorbate 1-(2-Hydroxyethyl)-4,6-Dimethyl-1,2-Dihydropyrimidine-2-One Upon Exposure to Carbon Tetrachloride on the basis Xymedon, were assessed in white rats exposed to CCl4. The compound under study administered prior

Elevated levels of proinflammatory cytokines in cerebrospinal fluid of multiple sclerosis patients cytokines, including IL-2RA, CCL5, CCL11, MIF, CXCL1, CXCL10, IFNγ, SCF, and TRAIL, were upregulated

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