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Immunohistochemical study of Ki-67, PHH3, and CK15 protein expression in oral epithelial malignancy in situ and squamous cell carcinoma. © A.A. IVINA, V.A. SEMKIN, Z.S. KHABADZE, I.I. BABICHENKO.

Value of the expression of Ki-67 and membrane protein glucose transporter GLUT1 in the diagnosis of neoplastic transformation of the oral mucosal epithelium.6%) had squamous cell carcinoma. Ten intact MO samples were used as a control group. Tissue antigens were

Clinical and morphological features of oral leukoplakia gradation systems of oral epithelial dysplasiaand a new term «differentiated dysplasia» in leukoplakia. A

Cytokeratin 15 as a diagnostic marker for oral epithelial malignization [Tsitokeratin 15 kak diagnosticheskiĭ marker nachala malignizatsii épiteliia slizistoĭ obolochki rta] intraepithelial neoplasia may be a diagnostic criterion for the beginning of oral epithelial malignization

The role of Sonic Hedgehog signaling pathway in the development of oral squamous cell carcinoma carcinoma. Tissue antigens were determined using a mouse monoclonal antibody to Ki-67 protein and rabbit

Morphology of epithelial-mesenchymal transformation in neoplasias of oral mucosa [Morfologicheskie osnovy épitelial'no-mezenkhimal'noĭ transformatsii épiteliia slizistoĭ obolochki rta pri neoplazii] determined using a mouse monoclonal antibody to Ki-67 and E-cadherin and rabbit polyclonal antibodies to N

Molecular genetic criteria for oral mucosal epithelial malignization by FISH in Group 1 was almost absent and showed a 2:2 signal ratio in 1.3% of cases; in other groups

Immunohistochemical criteria for differential diagnostics of squamous epithelium changes in pyogenic granuloma and squamous cell carcinoma of the oral mucosa

Modern perspectives of oral squamous cell carcinoma [Sovremennye predstavleniya o ploskokletochnom rake slizistoi obolochki rta], syphilitic glossitis and actinic keratosis (liponly). A binary system for assessing oral epithelial dysplasia

Papillomavirus in the genesis of oral leukoplakia infection involving P16INK4a and HPV16 proteins in different cell areas of the intact mucosa, in leukoplakia

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