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Архитектура системы контроля доступа LPS защищенной ос BastionАрхитектура системы контроля доступа LPS защищенной ос Bastion

Dual Effect of Low-Molecular-Weight Bioregulators of Bacterial Origin in Experimental Model of Asthma (LPS) per animal for 5 days before sensitization by ovalbumin (OVA). Preliminary administration of LPS

Ammonia Attenuates LPS-Induced Upregulation of Pro-Inflammatory Cytokine mRNA in Co-Cultured Astrocytes and Microglia-cultured astrocytes and microglia. In mono-cultured microglia LPS (100 ng/ml, 18 h) strongly elevated mRNA levels

Oxylipin profiles as functional characteristics of acute inflammatory responses in astrocytes pre-treated with IL-4, IL-10, or LPS treated for 24 h with lipopolysaccharide (LPS) for classical pro-inflammatory adaptation

Dependence of the severity of the systemic inflammatory response on resistance to hypoxia in male wistar rats response induced by lipopolysaccharide (LPS) in animals with different resistance levels to hypoxia

Bacterial agonists of innate immunity LPS regulates spontaneous and induced production of alfa defensins of human neutrophils in vitroBacterial agonists of innate immunity LPS regulates spontaneous and induced production of alfa

Change in the functional state of cells of the human endothelial cell line ECV-304 under the influence of influenza a virus, lipopolysaccharide from Escherichia coli and some drugsInfluenza A virus and bacterial lipopolysaccharide (LPS) cause dysfunction of the vascular

Inflammation Regulation by Bacterial Molecular Patterns-inflammatory cytokine TNF-α under the influence of TLR4 and NOD2 agonists, lipopolysaccharide (LPS

Sex-mediated differences in lps induced alterations of TNFα, IL-10 expression, and prostaglandin synthesis in primary astrocytes examined whether male and female primary rat astrocytes show different responses to lipopolysaccharide (LPS

Inhaled aerosolised recombinant human activated protein C ameliorates endotoxin-induced lung injury in anaesthetised sheep protein C (APC) attenuates ovine lipopolysaccharide (LPS)-induced lung injury. In this study, our aim

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