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Editorial: Cardio-Protection and Heart Repair: New Drugs, Targets and Approaches
Zagidullin, N., Cai, B., Zhu, H.
ischemic/reperfusion injury
Correction of morphofunctional disorders with asialoerythropoietin and selective inhibitor of arginase II KUD975 in cases of ischemic kidney damage in the experiment
Elagin, V. V., Bratchikov, O. I., Zatolokina, M. A.
Acute kidney injury (AKI), which is based on ischemic-reperfusion damage, is a widespread life
2-aminoaethanesulfonic acid compounds possess protective property in reperfusion-induced heart jnjury
Kurganov, N. A., Blinova, E. V., Semeleva, E. V., Gromova, I. A., Blinov, D. S.
in reperfusion-induced heart injury. The study was performed on rats and dogs of both sexes, isolated rats
Human recombinant erythropoietin gradient dosage influence on ischemic and reperfusion liver injury
Alehin, S. A., Kolmykov, D. I., Pokrovskii, M. V.
velocity and morphological changes in liver during ischemic and reperfusion injury. It was proved what
Metabolic cardioprotection: new concepts in implementation of cardioprotective effects of meldonium
Danilenko, L. M., Klochkova, G. N., Kizilova, I. V., Korokin, M. V.
Recent studies confirm the need to find means to correct ischemic / reperfusion injury due
Assessment of the nephroprotective properties of the erythropoietin mimetic peptide and infliximab in kidney ischemia-reperfusion injury in rat
Netrebenko, A. S., Gureev, V. V., Pokrovskii, M. V., Gureeva, A. V., Tsuverkalova, Y. M., Rozhkov, I. S.
HBSP) and infliximab on ischemic renal reperfusion injur. The experiment was performed on 70 white male Wistar
Tadalafil as an agent of pharmacological preconditioning in ischemic - reperfusion brain injury
Martynova, O. V.
pharmacological agents that can reduce the degree of ischemic-reperfusion injury of the brain, remains one
Human recombinant erythropoietin gradient dosage influence on ischemic and reperfusion liver injury
Alehin, S. A., Kolmykov, D. I., Pokrovskii, M. V.
velocity and morphological changes in liver during ischemic and reperfusion injury. It was proved what
c-Jun N-terminal kinases (JNKs) in myocardial and cerebral ischemia/reperfusion injury
Shvedova M., Anfinogenova Y., Atochina-Vasserman E., Schepetkin I., Atochin D.
ischemia/reperfusion injury. Numerous studies demonstrate that JNK-mediated signaling pathways play
Correction of retinal ischemic injuries by using non-selective imidazoline receptor agonists in the experiment
Levkova, E. A., Pazhinsky, A. l., Lugovskoy, S. S., Peresypkina, A. A., Bashuk, V. V.
with C7070 on the retinal ischemia-reperfusion model in rats

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