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Acid Sensitive Ion Channels as Target of Hydrogen Sulfide in Rat Trigeminal Neurons-sodium hydrogen sulfide (NaHS) on the firing of trigeminal (TG) nerve was explored using suction electrode
Hunting for origins of migraine pain: Cluster analysis of spontaneous and capsaicin-induced firing in meningeal trigeminal nerve fibers and Giniatullin. Trigeminal nerves in meninges are implicated in generation of nociceptive firing underlying
Acid Sensitive Ion Channels as Target of Hydrogen Sulfide in Rat Trigeminal Neurons-sodium hydrogen sulfide (NaHS) on the firing of trigeminal (TG) nerve was explored using suction electrode
Cortical spreading depression induces oxidative stress in the trigeminal nociceptive system with migraine aura, and tested nociceptive firing triggered by ROS in trigeminal nerves ex vivo. Application
Hunting for origins of migraine pain: Cluster analysis of spontaneous and capsaicin-induced firing in meningeal trigeminal nerve fibers and Giniatullin. Trigeminal nerves in meninges are implicated in generation of nociceptive firing underlying
Protective Effects of Hydrogen Sulfide Against the ATP-Induced Meningeal Nociception of trigeminal nerve activity in meninges was supplemented by patch-clamp and calcium imaging studies of isolated
Antidromic Spike Propagation and Dissimilar Expression of P2X, 5-HT, and TRPV1 Channels in Peripheral vs. Central Sensory Axons in MeningesBackground: The terminal branches of the trigeminal nerve in meninges are supposed to be the origin
EVALUATION OF THE EFFECTIVENESS OF THE METHOD OF STAINING AND STORAGE OF ANATOMIC PREPARATIONS CONTAINING THIN NERVES life of museum and demonstration drugs, for a more intense coloring of the intra- and extra-nerve
Proliferative potencies of the thalamus nerve cells in tadpoles and adult frogs Xenopus laevis after its injury (Russian) neurons of the type of neurons of the trigeminal nerve mesencephalic nucleus and the large ganglion cells
Deciphering in silico the Role of Mutated NaV 1.1 Sodium Channels in Enhancing Trigeminal Nociception in Familial Hemiplegic Migraine Type 3, at trigeminal nerve terminals. In WT Aδ-fibers of meningeal afferents, NaV1.1 channels efficiently participate
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