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SARS-CoV-2 Cell Entry Depends on ACE2 and TMPRSS2 and Is Blocked by a Clinically Proven Protease Inhibitor of coronaviruses depends on binding of the viral spike (S) proteins to cellular receptors and on S protein priming
Molecular mimicry between SARS-CoV-2 spike glycoprotein and mammalian proteomes: implications for the vaccineMolecular mimicry between SARS-CoV-2 spike glycoprotein and mammalian proteomes: implications
Molecular Targets in the Chemotherapy of Coronavirus Infection. In the first stage, when the virus multiplication and accumulation are prevalent under insufficient host immune
Photopolarimetrical properties of coronavirus model particles: Spike proteins number influence the degree of danger of the virus. A larger number of spike proteins makes the virus infectivity stronger
Strategies for Targeting SARS CoV-2: Small Molecule Inhibitors—The Current StatusSevere Acute Respiratory Syndrome-Corona Virus-2 (SARS-CoV-2) induced Coronavirus Disease - 19
Nafamostat mesylate blocks activation of SARS-CoV-2: New treatment option for COVID-19virus spike protein
Intrinsic disorder perspective of an interplay between the renin-angiotensin-aldosterone system and SARS-CoV-2 involves interaction of the virus (via the receptor-binding domain (RBD) of its spike glycoprotein
Establishment of murine hybridoma cells producing antibodies against spike protein of sars‐cov‐2 mouse mAbs against receptor binding domain of SARS‐CoV‐2 spike (S) protein. These mAbs are able
Study of adsorption of the SARS-CoV-2 virus spike protein by vibrational spectroscopy using terahertz metamaterialsAdhesion of the spike protein of the SARS-CoV-2 virus is studied by vibrational spectroscopy using
Why covid-19 transmission is more efficient and aggressive than viral transmission in previous coronavirus epidemics?virus spike protein
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