Circulating blood cells and extracellular vesicles in acute cardioprotectionDavidson S.,
Andreadou I.,
Barile L.,
Birnbaum Y.,
Cabrera-Fuentes H.,
Cohen M.,
Downey J.,
Girao H.,
Pagliaro P.,
Penna C.,
Pernow J.,
Preissner K.,
Ferdinandy P. is essential to minimize cardiac
injury but can paradoxically cause further damage. Experimental procedures
Translational research in acute lung injury and pulmonary fibrosis attenuating endogenous Fgfr2b ligands during bleomycin-induced lung fibrosis does not compromise murine lung repairMackenzie B.,
Henneke I.,
Hezel S.,
Alam D.,
Agha E.,
Chao C.,
Quantius J.,
Wilhelm J.,
Jones M.,
Goth K.,
Li X.,
Seeger W.,
Königshoff M.,
Herold S.,
Rizvanov A.,
Günther A.,
Bellusci S. , receptors, and signaling targets in wild-
type mice following bleomycin lung
injury. In addition, the impact
Regeneration of rat sciatic nerve using tubulation and allogeneic transplantation of adipose-derived mesenchymal stromal cellsMasgutov R.,
Masgutova G.,
Rogojin A.,
Zhuravleva M.,
Zakirova E.,
Nigmetzyanova M.,
Mukhametova L.,
Shulman A.,
Mukhamedshina Y.,
Yafarova G.,
Rizvanov A. ) on posttraumatic peripheral nerve regeneration was investigated using a rat model of sciatic nerve
injury. We
СПОСОБ КОНТРОЛЯ ЭФФЕКТИВНОСТИ ГЕМОСТАЗА ПРИ ТРАВМАТИЧЕСКИХ ПОВРЕЖДЕНИЯХ ОРГАНОВ И СТРУКТУР ЗАБРЮШИННОГО ПРОСТРАНСТВАТИМЕРБУЛАТОВ, В.М.,
ГАРЕЕВ, Р.Н.,
ФАЯЗОВ, Р.Р.,
ТИМЕРБУЛАТОВ, Ш.В.,
Timerbulatov, V.M.,
Gareev, R.N.,
Fayazov, R.R.,
Timerbulatov, Sh.V. METHOD FOR MONITORING EFFECTIVENESS OF HEMOSTASIS IN TRAUMATIC
INJURIES OF ORGANS AND
STRUCTURES The concurrence of kidney and liver dysfunctions in decompensated heart failure in 87%, myocardial infarction in 57%, atrial fibrillation in 65%, chronic kidney disease in 39%,
type 2
Targeting inerleukin-6 for renoprotection-signaling) thus affecting distinct cell
types and eliciting various metabolic, cytoprotective, or pro
ОСОБЕННОСТИ ЗАБРОНЕВОЙ ТРАВМЫ ПРИ ИСПОЛЬЗОВАНИИ СОВРЕМЕННЫХ БРОНЕЖИЛЕТОВ СКРЫТОГО НОШЕНИЯ of penetrating
injuries to the chest and abdomen even when the protective
structure is not broken. The volume
HtrA Protease from Bacillus subtilis Suppresses the Bacterial Fouling of the Rat Skin Injuries and Staphylococcus epidermidis. While the model rat skin
injuries treated with HtrA healed slower than in the case