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Overexpression of RPOTmp Being Targeted to Either Mitochondria or Chloroplasts in Arabidopsis Leads to Overall Transcriptome Changes and Faster Growth, targeted either to mitochondria or chloroplasts, disturbs the dormant seed state, and it causes

Mitochondrial destiny in type 2 diabetes: The effects of oxidative stress on the dynamics and biogenesis of mitochondria of mitochondria and mitochondrial DNA (mtDNA) in obesity and type 2 diabetes were analyzed. Results. Oxidative

Mitochondrial Dysfunctions May Be One of the Major Causative Factors Underlying Detrimental Effects of Benzalkonium Chloride in mitochondria and yeast cells was almost totally prevented by the mitochondria-targeted antioxidant SkQ1

Influence of AEDG and KE peptides on mitochondrial staining and the expression of ribosomal protein l7a with aging of the human pineal gland and thymus cell In vitroNew molecular targets for the geroprotective activity of AEDG (epitalon) and KE (vilon) peptides

Synthesis and biological evaluation of lipo-philic triphenylphosphonium derivatives of acetylsalicylic and salicylic acids as energy metabolism inhibitors in cellsmitochondria-targeted

Mitochondria Donation by Mesenchymal Stem Cells: Current Understanding and Mitochondria Transplantation StrategiesThe phenomenon of mitochondria donation is found in various tissues of humans and animals

Mitochondria-targeted mesoporous silica nanoparticles noncovalently modified with triphenylphosphonium cation: Physicochemical characteristics, cytotoxicity and intracellular uptake cation that improves targeted delivery of loads to mitochondria.

Calcium signalling in sensory neurones and peripheral glia in the context of diabetic neuropathies responsible for Ca2+ homeostasis and signalling, therefore, can be regarded as potential therapeutic targets.

Mitochondrial DNA Mutations in Colorectal Cancer Stem Cells: Implications for Tumor Dynamics and Therapeutic Strategies of mtDNA mutations. The review hypothesizes that CSCs rely on well-functioning mitochondria for crucial

β-adrenergic over-stimulation and cardio-myocyte apoptosis: Two receptors, one organelle, two fates? as inducing apoptosis of cardio-myocytes. At the same time as being the main oxygen consumers, mitochondria

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