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Interaction of immune response mediator genes in a predisposition to juvenile idiopathic arthritis the polymorphic loci of immune response mediator genes (TNFA rs1800629, LTA rs909253, IL1B rs16944, IL2-IL21 rs

INTERLEUKIN GENE POLYMORPHISM AND RISK OF UTERINE FIBROIDS polymorphic loci of interleukin genes were selected for the study, namely A/C IL10 rs1800872, C/G IL6 rs

Contribution of IL12A, IL12B, IL13 and IL12RB2 gene polymorphisms to the development of chronic obstructive pulmonary disease in the ethnic group of tatarsПроведен анализ ассоциации полиморфных вариантов генов IL12A (rs568408, rs2243115), IL12B (rs

Candidate SNP markers of aggressiveness-related complications and comorbidities of genetic diseases are predicted by a significant change in the affinity of TATA-binding protein for human gene promoters computer-based search methods for SNPs (that alter gene expression) (i) Web service SNP

FGB, TNFα, IL-1β, LPL, ITGB3, and TGFB1 genes polymorphism in patients with recurrent myocardial infarction people with recurrent MI compared to 280 people who had just one episode of MI. TNFα (rs1800629), IL1B

IL-28B gene polymorphism predicts the response to antiviral treatment in chronic hepatitis C in particular IL-28B gene polymorphism.

INVESTIGATION OF THE ROLE OF CYTOKINE GENES POLYMORPHISMS IN THE DEVELOPMENT OF THE URTICARIA IN THE REPUBLIC OF BASHKORTOSTAN of interleukins genes IL4 (rs2243250), IL4R (rs1805010), IL10 (rs1800872), IL13 (rs20541) and tumor necrosis

Expression of immune, antioxidant and stress related genes in different organs of common carp exposed to indoxacarb of indoxacarb increased inflammatory cytokine gene expression (IL-1β, IL-8, IL-10, TNF-α and IFN-γ) and inhibits

Obesity-related known and candidate SNP markers can significantly change affinity of TATA-binding protein for human gene promoters of obesity. Near the sites binding to TATA-binding protein (TBP) in human gene promoters, we found 22 obesity

ASSOCIATION OF GENE POLYMORPHISMS WITH RECURRENT SPONTANEOUS ABORTIONGene polymorphisms and abnormal gene interactions may be related to the idiopathic etiology of RSA

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