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Sensitivity of acute myeloid leukemia Kasumi-1 cells to binase toxic action depends on the expression of KIT and AML1-ETO oncogenes human KIT N822K oncogene and/or human AML1-ETO oncogene and human leukemia Kasumi-1 cells expressing
Sensitivity of acute myeloid leukemia Kasumi-1 cells to binase toxic action depends on the expression of KIT and AML1-ETO oncogenes human KIT N822K oncogene and/or human AML1-ETO oncogene and human leukemia Kasumi-1 cells expressing
Germline Variants in Proto-Oncogenes and Tumor Suppressor Genes in Women with Cervical Cancer pathogenesis. This study aimed to investigate germline variants in proto-oncogenes and tumor suppressor genes
PTPN11 Knockdown Prevents Changes in the Expression of Genes Controlling Cell Cycle, Chemotherapy Resistance, and Oncogene-Induced Senescence in Human Thyroid Cells Overexpressing BRAF V600E Oncogenic Protein) involved in cell cycle regulation and oncogene-induced senescence in response to BRAF V600E expression
RET/PTC3 Rearrangement in Papillary Thyroid Carcinoma: Possible Marker of Tumor ProgressionProto-Oncogene Proteins
RAB42: A Prognostic Marker Associated with Tumor Immune Microenvironment in Bladder CancerRAB42, member rat sarcoma oncogene family was proved to play a key role in cancer
Viral Infections: Negative Regulators of Apoptosis and Oncogenic Factors. Oncogenic viruses manipulate both external and internal apoptosis pathways, and inhibit the activity
HYPERPLASTIC PROCESSES IN THE CERVICAL CANAL OF PATIENTS WITH HIGH-RISK OR ONCOGENIC HUMAN PAPILLOMAVIRUSHYPERPLASTIC PROCESSES IN THE CERVICAL CANAL OF PATIENTS WITH HIGH-RISK OR ONCOGENIC HUMAN
Oncogenic c-kit transcript is a target for binase to binase is caused by the expression of c-kit oncogene, the cytotoxicity of the RNase was studied
Genetical mechanisms of viral oncogenesis of viral and cellular oncogenes and mechanisms of their action. Structures of genomes of main groups
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