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Alterations in lipid mediators of inflammation associated with glaucomatous damage in a rabbit model

Mechanisms of glaucoma: the insights from lipidomic studies

Mechanisms of ocular surface inflammation associated with iatrogenic damage to the cornea

Therapeutic proteins for treatment of corneal epithelial defects

Inflammatory metabolites of arahidonic acid in tear fluid in UV-induced corneal damage of arachidonic acid and prostaglandin E2 and an increase in the concentrations of prostaglandin D2 and its

Inflammatory Metabolites of Arachidonic Acid in Tear Fluid in UV-Induced Corneal Damage, including a decrease in the amount of arachidonic acid and prostaglandin E2 and an increase

Comparative lipidomic analysis of inflammatory mediators in the aqueous humor and tear fluid of humans and rabbits

Targeted Lipidomic Analysis of Aqueous Humor Reveals Signaling Lipid-Mediated Pathways in Primary Open-Angle Glaucoma

Alterations in Tear Content of Inflammatory Oxylipines Associated with Perioperative Dry Eye Syndrome of PDES. The majority of metabolites of LA (13-HODE and 9-HODE), ALA (9-HOTrE and 13-HOTrE) and AA (12

Tear Fluid Inflammatory Oxylipins in Perioperative Dry Eye Disease and 9-HODE), ALA (9-HOTrE and 13-HOTrE) and AK (12-HETE), the tear content of which is changed in PDES

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