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Regulation of acetylcholinesterase activity by nitric oxide in rat neuromuscular junction via N-methyl-d-aspartate receptor activation-mediator of acetylcholine, is capable of triggering the NMDA receptor/NO synthase-mediated pathway that modulates synaptic

The Degree of Oxidative Stress in the Rat Brain During Ischemia and Reperfusion in Conditions of Correction of the L-Arginine-No System NO synthase activity was responsible in the late period.

Complex nucleotide medicine from Saccharomyces cerevisiae cells - activator of Ca2+ -dependent NO-synthase. The activation effect of the nucleotide preparation on Ca2+-dependent NO-synthase was evaluated as compared

ANALYSIS ON THE INFLUENCE OF VARIOUS SUBSTANCES ON SELECTIVE ACTIVATION OF CA2+ -DEPENDENT NO-SYNTASE (REVIEW)The article presents information on the structure, classification and functions of NO-synthases

Regulation of acetylcholinesterase activity by nitric oxide in rat neuromuscular junction via N-methyl-d-aspartate receptor activation-mediator of acetylcholine, is capable of triggering the NMDA receptor/NO synthase-mediated pathway that modulates synaptic

Effect of NO Synthase Blockade on NO Production in Rat Heart under Conditions of Hypokinesia that the increase in NO production during hypokinesia is associated with activation of NO synthases.

Participation of NO-synthase system in the stress-mediated reactions of the brain. Expression of neuronal constitutive (nNOS) and inducible (iNOS) isoforms of NO-synthase was studied

Evaluation of anti-inflammatory activity of experimental samples of medicinal forms based on liquid plant extracts on inducible NO-Synthase (iNOS). The results obtained show an effective anti-inflammatory activity of tablets

Toward adhesion-free endoscopy?[No abstract available]

The State of the Nitric Oxide Cycle in Respiratory Tract Diseases nitric oxide cycle has been expanded to include the NO-synthase and NO-synthase-independent component

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