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Dynamics of endothelial function indexes in patients with post-Covid syndrome using a combination drug of ethylmethylhydroxyperidine succinate/vitamin B6Aim: to evaluate endothelial function in patients after COVID-19 before and after treatment

Studying dose-dependent endothelio- and cardioprotective activity of selective arginase II inhibitor in hyperhomocysteineinduced endothelial dysfunction-induced endothelial dysfunction

Down-regulation of inducible nitric oxide synthase (iNOS) in rat with congenital hydronephrosis-arginine by nitric oxide synthase, and in the kidney iNOS is expressed spontaneously. The aim of our study

EPR study of nitric oxide production in rat tissues under hypokinesiaEPR spectroscopy was used to study the intensity of nitric oxide (NO) production upon modeling 60

Endothelial dysfunction: comparative evaluation of ultrasound dopplerography, laser dopplerflowmetry and direct monitoring of arterial pressure for conducting pharmacological tests in rats and with the blockade of the nitric oxide synthesis by means of the ultrasound method for examining the central blood

The cardio- and endothelial protective effects of ethyl methyl hydroxyl pyridine malate in modeling L-name induced nitric oxide deficiencyCurrently, endothelial dysfunction is considered as a predictor of a number of pathologies

Influence of Nonspecific Inhibitor of NO-Synthase L-NAME on Electric Characteristics of Premotor Interneurons of Terrestrial Snails to hyperpolarization shift of the membrane potential caused by the action of the nitric oxide donor

Effects of NO Donors and Inhibitors of NO Synthase and Guanylate Cyclase on the Acquisition of a Conditioned Defense Food Aversion Response in Edible Snails that NO synthase blockade with a nonspecific neuronal NO synthase inhibitor L-NAME before defense food aversion

Effect of NO Synthase Blockade on Myocardial Contractility of Hypokinetic Rats during Stimulation of β-Adrenoreceptors-selective blocker of NO synthases) decreased or increased myocardial contractility, respectively. In control rats

Regulation of acetylcholinesterase activity by nitric oxide in rat neuromuscular junction via N-methyl-d-aspartate receptor activation and glycine led to enhancement of nitric oxide (NO) production, resulting in partial AChE inhibition. Partial

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