Non-mouse-adapted H1N1pdm09 virus as a model for influenza research to reach the pathogenicity level of the classical
mouse-adapted PR8 virus. © 2020 by the authors.
Establishment of murine hybridoma cells producing antibodies against spike protein of sars‐cov‐2 mouse mAbs against receptor binding domain of SARS‐CoV‐2 spike (S) protein. These mAbs are able
Adaptation of pandemic H1N1 influenza viruses in mice by promoting their
mouse adaptation. The resulting
mouse lung-adapted variants showed significantly enhanced
Septin dynamics are essential for exocytosis-regulation of septin-5 in various cells and
mouse models or septin-4 in mice suggested either an inhibitory or a