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Evaluation of methods of modeling and formation of experimental allergic encephalomyelitisExperimental autoimmune (allergic) encephalomyelitis (EAE) induced by intradermal injection

Ubiquitin-independent proteosomal degradation of myelin basic protein contributes to development of neurodegenerative autoimmunity to the destruction of axons. Here, we show that brainderived proteasomes from SJL mice with experimental autoimmune

Production of abzymes in th, cba, and c57bl/6 mice before and after mog treatment: Comparing changes in cell differentiation and proliferationTill yet there is no data concerning mechanisms of autoimmune diseases development. Experimental

Ubiquitin-independent proteosomal degradation of myelin basic protein contributes to development of neurodegenerative autoimmunity to the destruction of axons. Here, we show that brainderived proteasomes from SJL mice with experimental autoimmune

Changes in cell differentiation and proliferation lead to production of abzymes in EAE mice treated with DNA–Histone complexesExperimental autoimmune encephalomyelitis (EAE)-prone C57BL/6 mice are used as a model of human

Catalytic antibodies in the bone marrow and other organs of Th mice during spontaneous development of experimental autoimmune encephalomyelitis associated with cell differentiation of autoimmune reaction development in experimental autoimmune encephalomyelitis (EAE) prone Th mice

Changes in different parameters, lymphocyte proliferation and hematopoietic progenitor colony formation in EAE mice treated with myelin oligodendrocyte glycoprotein used MOG35-55-induced experimental autoimmune encephalomyelitis (EAE), a model of human MS, to assess

Changes in haematopoietic progenitor colony differentiation and proliferation and the production of different abzymes in EAE mice treated with DNAImmunization of experimental autoimmune encephalomyelitis (EAE)-prone C57BL/6 mice with MOG35-55 (a

Intravenous immunoglobulin in treatment of autoimmune neurological diseases in children, these drugs are widely used in treatment of autoimmune diseases. In this review, we have analyzed

Specific depletion of myelin-reactive B cells via BCR-targeting cells in mice with experimental autoimmune encephalomyelitis. The proposed conception may be further

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