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Modelling of the Innate and Adaptive Immune Response to SARS Viral Infection, Cytokine Storm and VaccinationIn this work, we develop mathematical models of the immune response to respiratory viral infection

Immuno-epidemiological model of two-stage epidemic growth stage of epidemic where only people with weak immune response are infected to the second stage where

The Influence of Immune Response on Spreading of Viral Infection the influence of the immune response on the infection spreading speed and on the viral load characterizing

The Influence of Immune Response on Spreading of Viral Infection by the innate immune response mostly determined by the local interferon production. The modeling results show

STABILITY ANALYSIS OF A VIRAL IMMUNE RESPONSE MODEL INVOLVING TWO TIME DELAYS differential equation arising from an immune response mathematical model. We use the monotone dynamical systems

The impact of immune cell interactions on virus quasi-species formation virus replication within host cells, transportation, and the immune response. Reaction-diffusion systems

MODELING IMMUNODOMINANCE IN THE B-CELL RESPONSE TO VIRAL INFECTIONAdaptive immune response can be accompanied by immunodominance which manifests itself

Stability Analysis of a Delayed Immune Response Model to Viral Infection typical forms of the function f, specifying the efficiency of immune response-mediated virus elimination

Bistability and damped oscillations in the homogeneous model of viral infection; [Бистабильность и затухающие колебанияв гомогенной модели вирусной инфекции] regimes of infection progression, we analyze the general mathematical model of immune response to viral

Mathematical modeling reveals that the administration of EGF can promote the elimination of lymph node metastases by PD-1/PD-L1 blockade between tumor progression and the immune response, we develop two new models describing the interaction

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