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Apoptotic endonuclease EndoG induces alternative splicing of telomerase catalytic subunit hTERT and death of tumor cellsTelomerase activity is known to be regulated by alternative splicing of its catalytic subunit h

Apoptotic endonuclease endog regulates alternative splicing of human telomerase catalytic subunit htertHuman telomerase catalytic subunit hTERT is subjected to alternative splicing results in loss

Inhibition of telomerase activity by splice-switching oligonucleotides targeting the mRNA of the telomerase catalytic subunit affects proliferation of human CD4+ T lymphocytesTelomerase activity is regulated at the mRNA level by alternative splicing (AS) of its catalytic

A link between alternative splicing and nonsense-mediated mRNA decay in the control of THIC gene expression in arabidopsis) encodes two mRNAs produced by alternative splicing; the ratio between these mRNAs is controlled

Intracellular localization of apoptotic endonuclease EndoG and splice-variants of telomerase catalytic subunit hTERT be regulated by alternative splicing of its mRNA. The mechanism of hTERT splicing is not understood in detail

Inhibition of nuclease activity by a splice-switching oligonucleotide targeting deoxyribonuclease 1 mRNA prevents apoptosis progression and prolong viability of normal human CD4+ T-lymphocytesThe nuclease activity of deoxyribonuclease 1 (DNase I) is regulated by alternative splicing (AS

Cytoprotective Activity of Polyamines Is Associated with the Alternative Splicing of RAD51A Pre-mRNA in Normal Human CD4+ T Lymphocytes. However, in normal cells, putrescin pretreatment resulted in alternative splicing of RAD51A and the switch

A link between alternative splicing and nonsense-mediated mRNA decay in the control of THIC gene expression in arabidopsis) encodes two mRNAs produced by alternative splicing; the ratio between these mRNAs is controlled

Induction of Telomerase Catalytic Subunit Alternative Splicing by Apoptotic Endonuclease G in Mouse and Rat LymphocytesIt is known that apoptotic endonuclease G (EndoG) induces alternative splicing (AS) of telomerase

Inhibition of Caspase-2 Activity in Human Jurkat T-cell Lymphoma Cells by Splice Switching Oligonucleotide to its pre-mRNA by alternative splicing (AS) of its mRNA. The aim of this work was to determine the ability of an oligonucleotide

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