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c-Jun N-terminal kinases (JNKs) in myocardial and cerebral ischemia/reperfusion injury ischemia/reperfusion injury. Numerous studies demonstrate that JNK-mediated signaling pathways play

The Degree of Oxidative Stress in the Rat Brain During Ischemia and Reperfusion in Conditions of Correction of the L-Arginine-No Systembrain, ischemia/reperfusion, oxidative stress, nitric oxide.

Tadalafil as an agent of pharmacological preconditioning in ischemic - reperfusion brain injury stroke and other modifications of brain ischemia. Therefore, the development and introduction of new

The Role of microRNAs in Epigenetic Regulation of Signaling Pathways in Neurological Pathologies the development of pathologies such as Alzheimer’s disease, cerebral ischemia and reperfusion injury

Influence of Pharmacological Product on the Basis Of Hopantenic Acid, Succinic Acid and Chitosan on Rats Tissues Free-Radical Homeostasis under Brain Ischemia/Reperfusion ischemia/ reperfusion (BIR). Males of white rats with weight of 150-200 g were used in experiments. BIR

Selenium in ischemic strokeSelenium plays a significant role in brain physiology, whereas data on the role of selenium

Human recombinant erythropoietin gradient dosage influence on ischemic and reperfusion liver injury velocity and morphological changes in liver during ischemic and reperfusion injury. It was proved what

Pharmacological preconditioning by recombinant erythropoietin as the possibility of increasing the stability of tissue of the retina to reperfusion ischemia in experiment the stability of tissue of the retina to reperfusion ischemia in experiment

Dynamics of the hypoxia—induced tissue edema in the rat barrel cortex in vitro in brain slices under ischemia-like conditions remain elusive. Here, we explored the dynamics of brain

Ischemia/reperfusion effect on pancreatic volumetrical blood flow velocityWe investigate blood flow velocity at ischemia/reperfusion early stages. Transient hyperemia

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