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Immunopathology and immunopharmacotherapy of coronavirus disease 2019 (covid-19): Focus on interleukin 6
Nasonov E.L.
and may be a stage of COVID-19 progression. A premium is placed to interleukin-6 (IL-6) in the spectrum
Activated Macrophages of Monocytic Origin Predominantly Express Proinflammatory Cytokine Genes, whereas Kupffer Cells Predominantly Express Anti-Inflammatory Cytokine Genes
Lokhonina A., Elchaninov A., Fatkhudinov T., Makarov A., Arutyunyan I., Grinberg M., Glinkina V., Surovtsev V., Bolshakova G., Goldshtein D., Sukhikh G.
of gene expression, which is particularly noticeable for M1- and M2-associated genes. For instance
The role of Interleukin 6 inhibitors in therapy of severe COVID-19
Nasonov E.
and chemokines, leading to pathologic activation of innate and adaptive (Th1 and Th17 mediated) immunity
Interleukins in Cancer Biology: Their Heterogeneous Role
Yuzhalin Arseniy
Interleukins in Cancer Biology responds to the growing need for credible and up-to-date information
COVID-19 biobank: features of the cytokine profile [Биобанк COVID-19: особенности цитокинового профиля]
Sushentseva N.N.
K panel — a soluble CD40 ligand (sCD40L), interleukin-1α (IL-1α), interleukin-1β (IL-1β
Investigating viral interference between influenza a virus and human respiratory syncytial virus in a ferret model of infection
Chan K.F.
interference between hRSV and 2009 pandemic influenza A(H1N1) virus (A[H1N1]pdm09) in the ferret model
A public health perspective of aging: Do hyper-inflammatory syndromes such as COVID-19, SARS, ARDS, cytokine storm syndrome, and post-ICU syndrome accelerate short- And long-term inflammaging?
Bektas A.
interleukin 1
Interleukin-6 is a potential target for a correction of endothelial dysfunction associated with low-grade systemic inflammation
Kostina, D. A., Pokrovskaya, T. G., Beskhmelnitsyna, E. A.
Interleukin-6 is a potential target for a correction of endothelial dysfunction associated with low
Pro-inflammatory cytokine response of the fluid contents of spermatoceles and epididymal cysts
Kocak I., Dundar M., Yenisey C., Serter M., Gunaydin G.
of interleukin 6 (IL-6), interleukin 8 (IL-8) and tumour necrosis factor alpha (TNF-α) in fluids of patients
BOB.1 controls memory B-cell fate in the germinal center reaction
Levels M.J.
-tolerance and the initiation of autoimmune disease. Here we show that mRNA levels of the transcription regulator BOB.1

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