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Is up-regulation gene expression of the certain genes during the viral respiratory tract infection would have any influence in pathogenesis of the SAR-CoV-2 infection?leukocyte immunoglobulin like receptor A3

Fundamental basis of covid-19 pathogenesis [Najznačajnije osnove patogeneze covid-19] through the respiratory tract and interacts primarily with toll-like receptors (TLRs). The events in SARS

Immunoglobulins with non-canonical functions in inflammatory and autoimmune disease statesImmunoglobulins are known to combine various effector mechanisms of the adaptive and the innate

Strategies to Prevent SARS-CoV-2-Mediated Eosinophilic Disease in Association with COVID-19 Vaccination and InfectionA vaccine to protect against COVID-19 is urgently needed. Such a vaccine should efficiently induce

β-endorphin-like peptide SLTCLVKGFY is a selective agonist of nonopioid β-endorphin receptorIt has been found that β-endorphin (β-END) and a synthetic β-END-like decapeptide Ser

The influence of graft-versus-host disease prophylaxis on the donor natural killer cell alloreactivity after α/βTCR/CD19+-depleted allogeneic hematopoietic stem cell transplantation in pediatric patients with acute leukemia (killer cell immunoglobulinlike-receptors, KIRs) family receptors and NK-alloreactivity is based on a KIR

Construction and immunogenicity of a novel multivalent vaccine prototype based on conserved influenza virus antigens integrated into phage AP205 virus-like particles (VLPs). While VLPs containing the 3M2e alone induced

Corona (COVID-19) time musings: Our involvement in COVID-19 pathogenesis, diagnosis, treatment and vaccine planningimmunoglobulin M

Comparative analysis of B-Cell receptor repertoires induced by live yellow fever vaccine in young and middle-Age donors length and nearly error-free immunoglobulin profiling to compare plasma cell antibody repertoires

Innate and adaptive immunity in aging and longevity: The foundation of resilience-reactive protein, complement system, TLR/NF-κB, cGAS/STING/IFN 1,3 and AGEs/RAGE pathways, myeloid cells and NLRP3

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