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Amino Acid Substitution Patterns in the E6 and E7 Proteins of HPV Type 16: Phylogeography and Evolution

Zelenova E.E., Karlsen A.A., Avdoshina D.V., Kyuregyan K.K., Belikova M.G., Trotsenko I.D.

oncoproteins were downloaded from the NCBI GenBank database on October 6, 2022. Samples were filtered according

RET/PTC3 Rearrangement in Papillary Thyroid Carcinoma: Possible Marker of Tumor Progression

oncoprotein

Binase treatment increases interferon sensitivity and apoptosis in SiHa cervical carcinoma cells by downregulating E6 and E7 human papilloma virus oncoproteins

-treated SiHa cells showed reduced expression of E6 and E7 viral oncoproteins and increased expression

CagA-mediated pathogenesis of helicobacter pylori

island (cagPAI). CagA is also known as the first bacterial onco-protein, ranking the H. pylori

Human dendritic cells transfected with a human papilloma virus-18 construct display decreased mobility and upregulated cytokine production

Khaiboullina S., Morzunov S., Hall M., De Meirleir K., Rizvanov A., Lombardi V.

and upregulated E6 and E7 oncoprotein transcripts as detected by RT-PCR. Transfection of DCs also resulted in a

Human dendritic cells transfected with a human papilloma virus-18 construct display decreased mobility and upregulated cytokine production

Khaiboullina S., Morzunov S., Hall M., De Meirleir K., Rizvanov A., Lombardi V.

and upregulated E6 and E7 oncoprotein transcripts as detected by RT-PCR. Transfection of DCs also resulted in a

Oncogenic c-kit transcript is a target for binase

Mitkevich V., Petrushanko I., Kretova O., Spirin P., Zelenikhin P., Prassolov V., Tchurikov N., Ilinskaya O., Makarov A.

-Kit oncoprotein, we demonstrate that the elimination of c-Kit is the key factor in selective cytotoxicity

Oncogenic c-kit transcript is a target for binase

Mitkevich V., Petrushanko I., Kretova O., Spirin P., Zelenikhin P., Prassolov V., Tchurikov N., Ilinskaya O., Makarov A.

-Kit oncoprotein, we demonstrate that the elimination of c-Kit is the key factor in selective cytotoxicity

Screening of conditionally reprogrammed patient-derived carcinoma cells identifies ERCC3-MYC interactions as a target in pancreatic cancer-amplified PDX models and elicited rapid and profound depletion of the oncoprotein MYC, a transcriptional

Inhibitors of HSP90 function.

Zhdanov D.D., Kovalenko N.A.

oncoproteins function as a client proteins HSP90 and HSP90 in tumor cells possesses greater activity. Therefore

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